I have AS. This video is an extreme example of what can happen with this disease. It hurt my soul just to watch it. Most people (like me) just experience back pain, inflammation, and maybe some relatively mild fusion compared to what you see here.

I feel for the guy, and really happy to see his resolution.

Combine that with new research on how a majority of people who recover from COVID have lasting heart changes/inflammation/complications, and tell me again how eager you are to go to that party with all the proud non-mask-wearers: https://www.cidrap.umn.edu/news-perspective/2020/07/research-reveals-heart-complications-covid-19-patients

Know who you take advice from....
Just gonna leave this here....

Wiki-
Dr Michael Klaper-In August 1993 Klaper issued a medical certificate for the insurers of two movies that River Phoenix was working on at the time, Dark Blood and Interview With the Vampire. The certificate was signed by both Klaper and Phoenix and stated that Phoenix had never used "LSD, heroin, cocaine, alcohol in excess, or any other narcotic, depressant, stimulant or psychedelic. At the time Phoenix was deeply into the drug scene and died from a drug overdose two months later on October 31, 1993. Phoenix's death resulted in Dark Blood being scrapped, and his role in Interview with the Vampire being replaced by Christian Slater. A total $US5.7 million was paid out by the insurers of both movies as the result of Phoenix's death. Since Phoenix's death, insurance rates have increased significantly, medical certificates are no longer accepted, and actors are required to undergo more rigorous medical examination prior to being insured. [23]

According to oncologist David Gorski "Klaper subscribes to the all-too-common claim that a vegan diet is better than any other and supplements that claim with a belief that undergoing fasts, in which one consumes only water, is a major part of the path to health and wellness". He supports multiple pseudoscience medical claims such as acupuncture, chiropractic, naturopathy and border-line "germ theory denialism". Klaper also gives "highly dubious advice for cancer patients, even claiming that fasting can shrink malignant tumors. Klaper claims that fasts will clear up inflammation, eczema, arthritis and other issues. "The situation" according to Gorski, is "way more complicated than Dr. Klaper paints it". As a surgeon himself, Gorski is appalled that Klaper claims that fasting encourages "faster wound healing" a statement that Gorski calls "Bullshit!". Magician Penn Jillette reported on multiple podcasts that he has lost over 25 pounds on Klaper's water fast diet, Gorski responded that of course he will lose weight on a water-only diet. In Gorski's opinion as a medical doctor himself, "Jillette has fallen "hook, line, and sinker for a whole lot of dietary pseudoscience and promoting it on his show with a credulous interview with someone like Dr. Klaper". Gorski hopes that Jillette will eventually realize "that Dr. Klaper is peddling highly dubious claims (at best). Basically, the product Dr. Klaper is peddling in terms of science is a massive exaggeration based on dubious science, cherry picked cases, and bad evolutionary analogies. Worse, fasts, even when supervised by a physician, are potentially dangerous"

Bob thinks pneumonia = hemorrhagic fever...

Dude, it's an inflammation of the lungs caused by something else, like the flu or a cold. She just didn't take care of herself and suffers from no small amount of stress.

It's serious, for her, but you can't "catch" pneumonia.

It's pneumonia, not tuberculosis or hemorrhagic fever, Bob.

It's an inflammation of the lungs, not a disease in and of itself. The underlying cause could be as simple as the flu or a cold.

I thought the same thing every time she said inflammation but my favourite (eff off spellcheck) line comes at 6:45 ... "Glyphosate ... is perforating the holes in the gut".

My daughter is a grad student in biochemistry and says she only made it to the 2 minute mark before her head exploded.

TONS of things cure cancer. All day, every day. Doctors have no clue what cancer is. All they can do is cut, burn, or poison and cross their fingers.

I didn't say Cannabis was THE cure. It is A cure used by thousands with amazing efficacy. Everyone is different.

Here's 60+ studies for your perusal if you insist on the superiority of western scientific research:

"Cannabis, and the cannabinoid compounds found within it, has been shown through a large cannabisplantamount of scientific, peer-reviewed research to be effective at treating a wide variety of cancers, ranging from brain cancer to colon cancer. Below is a list of over 60 studies that demonstrate the vast anti-cancer properties of cannabis.
Studies showing cannabis may combat brain cancer:
Cannabidiol (CBD) inhibits the proliferation and invasion in U87-MG and T98G glioma cells. Study published in the Public Library of Science journal in October 2013.
Tetrahydrocannabinol (THC) can kill cancer cells by causing them to self-digest. Study published in the Journal of Clinical Investigation in September 2013.
CBD is a novel therapeutic target against glioblastoma. Study published in Cancer Research in March 2013.
Local delivery of cannabinoid-filled microparticles inhibits tumor growth in a model of glioblastoma multiforme. Study published in Public Library of Science in January 2013.
Cannabinoid action inhibits the growth of malignant human glioma U87MG cells. Study published in Oncology Reports in July 2012.
Cannabidiol enhances the inhibitory effects of THC on human glioblastoma cell proliferation and survival. Study published in the Molecular Cancer Therapeutics journal in January 2010.
Cannabinoid action induces autophagy-mediated cell death in human glioma cells. Study published in The Journal of Clinical Investigation in May 2009.
Cannabinoids inhibit glioma cell invasion by down-regulating matrix metalloproteinase-2 expression. Study published in Cancer Research in March 2008.
Cannabinoids and gliomas. Study published in Molecular Neurobiology in June 2007.
Cannabinoids inhibit gliomagenesis. Study published in the Journal of Biological Chemistry in March 2007.
A pilot clinical study of THC in patients with recurrent glioblastoma multiforme. The results were published in the British Journal of Cancer in June 2006.
Cannabidiol inhibits human glioma cell migration through an independent cannabinoid receptor mechanism. Study published in the British Journal of Pharmacology in April 2005.
Cannabinoids inhibit the vascular endothelial growth factor pathway (VEGF) in gliomas. Study published in the Journal of Cancer Research in August 2004.
Antitumor effects of cannabidiol, a nonpsychoactive cannabinoid, on human glioma cell lines. Study published in the Journal of Pharmacology in November 2003.
Inhibition of glioma growth in vivo by selective activation of the CB2 cannabinoid receptor. Study published in the Journal of Cancer Research in August 2001.
Studies showing cannabis may combat colorectal cancer:
Cannabigerol (CBG) can inhibit colon cancer cells. Study published in the Oxford journal Carcinogenesis in October 2014.
Inhibition of colon carcinogenesis by a standardised Cannabis Sativa extract with high content of CBD. Study published in Phytomedecine in December 2013.
Chemopreventive effect of the non-psychotropic phytocannabinoid CBD on colon cancer. Study published in the Journal of Molecular Medecine in August 2012.
Cannabinoids against intestinal inflammation and cancer. Study published in Pharmacology Research in August 2009.
Action of cannabinoid receptors on colorectal tumor growth. Study published by the Cancer Center of the University of Texas in July 2008.
Studies showing cannabis may combat blood cancer:
The effects of cannabidiol and its synergism with bortezomib in multiple myeloma cell lines. Study published in the International Journal of Cancer in December 2013.
Enhancing the activity of CBD and other cannabinoids against leukaemia. Study published in Anticancer Research in October 2013.
Cannabis extract treatment for terminal acute lymphoblastic leukemia of Philadelphia chromosome (Ph1). Study published in Case Reports in Oncology in September 2013.
Expression of type 1 and type 2 cannabinoid receptors in lymphoma. Study published in the International Journal of Cancer in June 2008.
Cannabinoid action in mantle cell lymphoma. Study published in Molecular Pharmacology in November 2006.
THC-induced apoptosis in Jurkat leukemia. Study published in Molecular Cancer Research in August 2006.
Targeting CB2 cannabinoid receptors as a novel therapy to treat malignant lymphoblastic disease. Study published in Blood American Society of Hemmatology in July 2002.
Studies showing cannabis can combat lung cancer:
Cannabinoids increase lung cancer cell lysis by lymphokine-activated killer cells via upregulation of Icam-1. Study published in Biochemical Pharmacology in July 2014.
Cannabinoids inhibit angiogenic capacities of endothelial cells via release of tissue inhibitor of matrix metalloproteinases-1 from lung cancer cells. Study published in Biochemical Pharmacology in June 2014.
COX-2 and PPAR-γ confer CBD-induced apoptosis of human lung cancer cells. Study published in Molecular Cancer Therapeutics in January 2013.
CBD inhibits lung cancer cell invasion and metastasis via intercellular adhesion molecule-1. Study published in the Journal of the Federation of American Societies for Experimental Biology in April 2012.
Cannabinoid receptors, CB1 and CB2, as novel targets for inhibition of non–small cell lung cancer growth and metastasis. Study published in Cancer Prevention Research in January 2011.
THC inhibits epithelial growth factor-induced (EGF) lung cancer cell migration in vitro as well as its growth and metastasis in vivo. Study published in the journal Oncogene in July 2007.
Studies showing cannabis may combat stomach cancer:
Cannabinoid receptor agonist as an alternative drug in 5-Fluorouracil-resistant gastric cancer cells. Study published in Anticancer Research in June 2013.
Antiproliferative mechanism of a cannabinoid agonist by cell cycle arrest in human gastric cancer cells. Study published in the Journal of Cellular Biochemistry in March 2011.
Studies showing cannabis may combat prostrate cancer:
Cannabinoids can treat prostate cancer. Study published by the National Institute of Health in October 2013.
Non-THC cannabinoids inhibit prostate carcinoma growth in vitro and in vivo: pro-apoptotic effects and underlying mechanisms. Study published in the British Journal of Pharmacology in December 2012.
The role of cannabinoids in prostate cancer: Basic science perspective and potential clinical applications. Study published in the Indian Journal of Urology in January 2012.
Induction of apoptosis by cannabinoids in prostate and colon cancer cells is phosphatase dependent. Study published in Anticancer Research in November 2011.
Studies showing cannabis may combat liver cancer:
Involvement of PPARγ in the antitumoral action of cannabinoids on hepatocellular carcinoma (CHC). Study published in Cell Death and Disease in May 2013.
Evaluation of anti-invasion effect of cannabinoids on human hepatocarcinoma cells. Study published on the site Informa Healthcare in February 2013.
Antitumoral action of cannabinoids on hepatocellular carcinoma. Study published in Cell Death and Differentiation in April 2011.
Studies showing cannabis may combat pancreatic cancer:
Cannabinoids inhibit energetic metabolism and induce autophagy in pancreatic cancer cells. Study published in Cell Death and Disease in June 2013.
Cannabinoids Induce apoptosis of pancreatic tumor cells. Study published in Cancer Research in July 2006.
Studies showing cannabis may combat skin cancer:
Cannabinoid receptor activiation can combat skin cancer. Study published by the National Institute of Health in October 2013.
Cannabinoids were found to reduce skin cancer by 90% in just 2 weeks. Study published in the Journal of Pharmacy and Pharmacology in July 2013.
Cannabinoid receptors as novel targets for the treatment of melanoma. Study published in the Journal of the Federation of American Societies for Experimental Biology in December 2006.
Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors. Study published in the Journal of Clinical Investigation, in January 2003.
Studies showing cannabis may combat other types of cancer:
Bladder: Marijuana reduces the risk of bladder cancer. Study published in the Medscape site in May 2013.
Kaposi sarcoma: Cannabidiol inhibits growth and induces programmed cell death in Kaposi sarcoma–associated herpesvirus-infected endothelium. Study published in the journal Genes & Cancer in July 2012.
Nose, mouth, throat, ear: Cannabinoids like THC inhibit cellular respiration of human oral cancer cells. Study by the Department of Pediatrics at the State University of New York, published in June 2010.
Bile duct: The dual effects of THC on cholangiocarcinoma cells: anti-invasion activity at low concentration and apoptosis induction at high concentration. Study published in Cancer Investigation in May 2010.
Ovaries: Cannabinoid receptors as a target for therapy of ovarian cancer. Study published on the American Association for Cancer Research website in 2006.
Preparation and characterisation of biodegradable microparticles filled with THC and their antitumor efficacy on cancer cell lines. Study published in the Journal of Drug Targeting in September 2013.
CBD Cannabidiol as a potential anticancer drug. Study published in the British Journal of Pharmacology in February 2013.
Cannabinoids as anticancer modulators. Study published in the Progress in Lipid Research journal in January 2013.
CBD inhibits angiogenesis by multiple mechanisms. Study published in the British Journal of Pharmacology in November 2012.
Towards the use of cannabinoids as antitumour agents. Study published in Nature in June 2012.
Cannabinoid-associated cell death mechanisms in tumor models. Study published in the International Journal of Oncology in May 2012.
Cannabinoids, endocannabinoids and cancer. Study published in Cancer Metastasis Reviews in December 2011.
The endocannabinoid system and cancer: therapeutic implication. Study published in the British Journal of Pharmacology in July 2011.
This list was compiled in part by Alchimiaweb.com.
– TheJointBlog"

For reference because this confuses the shit out of me.


Nonflammable -

non·flam·ma·ble
adjective \-ˈfla-mə-bəl\

: not burning or not burning easily : not easily set on fire
Full Definition of NONFLAMMABLE
: not flammable; specifically : not easily ignited and not burning rapidly if ignited

Flammable -



flam·ma·ble
[flam-uh-buhl] Show IPA
adjective
easily set on fire; combustible; inflammable.
Origin:
1805–15; < Latin flammā ( re ) to set on fire + -ble
inflammable -

in·flam·ma·ble
inˈflaməbəl/
adjective
adjective: inflammable

1.
easily set on fire.
"inflammable and poisonous gases"
synonyms: flammable, combustible, incendiary, ignitable;
volatile, unstable
"inflammable fabrics"
antonyms: fireproof


WHY NOTS THE ON FIRES OR NOT ON FIRES-ABLE ??

Per the study:
"60 of non-GM pigs had mild or moderate inflammation compared with 41 GM pigs, and only 4 non-GM pig stomachs were graded “nil,” while the GM pigs tallied up 8." And "The researchers found few statistically significant differences between the two groups after comparing them based on nearly 20 different parameters, including weight gain, stomach ulcers and kidney abnormalities. The GM-fed pigs did, however, show significantly higher rates of “severe” stomach inflammation, as well as an average of 25 percent heavier uteri in relation to body weight."

Also: "According to the study’s authors, the GM corn and soy used in the study were considered compositionally and substantially equivalent to the non-GM varieties by government agencies. But the lack of a controlled feed-growing environment potentially calls the results into question, according to Kent Bradford, Ph.D., director of the Seed Biotechnology Center at the University of California, Davis."

Lastly, the protection bill was actually an anti-litigation bill to prevent injunctions from tying up seed sales in court as has happened before.

Remember Kids, its only propaganda if the other guys do it, if we do it, its the truth!

It is generally understood that acetaminophen/paracetemol binds and reduces oxidized, 'highly active' COX-2 enzymes... this chills out the COX-2 activity until it is oxidized again and cranks back up. So imagine in this animation acetaminophen kind-of causing that active COX binding site to close up a little for a while, and preventing it from working in that way.

Acetaminophen also doesn't appear to have a lot of anti-inflammatory action near the actual site of inflammation, but rather more globally throughout the body; folks hypothesize a lot about the reason for this, but I like the simple answer that acetaminophen itself can't reduce oxidized COX enzymes in an injured area surrounded by high levels of oxidative species.

(nerd fist-bump)

>> ^MilkmanDan:

>> ^spoco2:
But what about paracetamol? That's different, because you can take the allowed dosage of that and the allowed dosage of ibuprofen at the same time without interaction... but how does it work?

Paracetemol is the major painkiller here in Thailand, which annoys me because I believe that I am essentially immune to it. I took paracetemol (tylenol) often when I was in my pre and early teens for migraines. At some point, I found that it just wouldn't do anything for me anymore. Ibuprofen and Aspirin both work great, so I always use those when I have a choice -- but the default is always Paracetemol here.

Rock isn't flammable

"Everyone then who hears these words of mine and acts on them will be like a wise man who built his house on rock. The rain fell, the floods came, and the winds blew and beat on that house, but it did not fall, because it had been founded on rock. And everyone who hears these words of mine and does not act on them will be like a foolish man who built his house on sand. The rain fell, and the floods came, and the winds blew and beat against that house, and it fell—and great was its fall!"

>> ^luxury_pie:
>> ^quantumushroom:
Shiny is here to 'sell' a point of view. Granted, it's the wrong audience to espouse fundamentalism, but if you see someone's house is burning in the night, you don't worry about their grumpiness at being awakened by total strangers.
Be grateful for hearing other points of view, and more grateful for resistance, it's the only thing that builds strength.
>> ^petpeeved:
>> ^quantumushroom:
Someone wrote of shiny: It's highly unlikely that you will ever convert anyone here but at the very least you'd be less despised if you weren't so angry and obnoxious all the time.
Shiny's the one being infinitely patient here, and by an act of free will endures these cheap shots. A less angry foe you do not have.
A good Christian spreads the Word, and in another Penn video Penn himself states if you Believe, you should be out spreading your message.
You should be grateful there are shinys out there to keep you honest, as honest as a liberal can be, anyway.

I can't swallow this. By its very nature, Christianity as espoused by Shiny is bigoted and infinitely intolerant (the ultimate end of impatience) of any view point in opposition of its literally written in stone beliefs.
Just because the evangelists adopt a cloyingly condescending tone that can be mistaken for politeness when they 'discuss' (read: lecture and don't listen) this with us heathens does not make them 'less angry' or 'infinitely patient'.


Generally speaking you are right. But in this case shiny is a supporter of the company which only builds houses out of highly inflammable compounds. And he may was at the crime scene, who knows.

>> ^quantumushroom:

Shiny is here to 'sell' a point of view. Granted, it's the wrong audience to espouse fundamentalism, but if you see someone's house is burning in the night, you don't worry about their grumpiness at being awakened by total strangers.
Be grateful for hearing other points of view, and more grateful for resistance, it's the only thing that builds strength.

>> ^petpeeved:
>> ^quantumushroom:
Someone wrote of shiny: It's highly unlikely that you will ever convert anyone here but at the very least you'd be less despised if you weren't so angry and obnoxious all the time.
Shiny's the one being infinitely patient here, and by an act of free will endures these cheap shots. A less angry foe you do not have.
A good Christian spreads the Word, and in another Penn video Penn himself states if you Believe, you should be out spreading your message.
You should be grateful there are shinys out there to keep you honest, as honest as a liberal can be, anyway.

I can't swallow this. By its very nature, Christianity as espoused by Shiny is bigoted and infinitely intolerant (the ultimate end of impatience) of any view point in opposition of its literally written in stone beliefs.
Just because the evangelists adopt a cloyingly condescending tone that can be mistaken for politeness when they 'discuss' (read: lecture and don't listen) this with us heathens does not make them 'less angry' or 'infinitely patient'.



Generally speaking you are right. But in this case shiny is a supporter of the company which only builds houses out of highly inflammable compounds. And he may was at the crime scene, who knows.

What I learned from this video: Inflammable = flammable.

@DrewNumberTwo:

She's not making it up. She's just not citing sources.

Five minutes of internet searching found me this:

"ACUTE TOXICITY

Chlorpropham is moderately toxic by ingestion (2). It may cause irritation of the eyes or skin (2). Symptoms of poisoning in laboratory animals have included listlessness, incoordination, nose bleeds, protruding eyes, bloody tears, difficulty in breathing, prostration, inability to urinate, high fevers, and death. Autopsies of animals have shown inflammation of the stomach and intestinal lining, congestion of the brain, lungs and other organs, and degenerative changes in the kidneys and liver (2)"

Breakdown of Chemical in Soil and Groundwater

Chlorpropham has some potential to contaminate groundwater because it is highly soluble in water and it has only a moderate tendency to adsorb to soil particles (3, 5). Chlorpropham adsorbs strongly to organic matter, so it is unlikely to leach through soils high in organic matter. Chlorpropham does not readily adsorb to montmorillonite or kaolinite clays (4).
Chlorpropham is subject to degradation by soil microbes. Photodegradation and volatilization do not readily occur. Increasing temperatures above 35 degrees C and increasing soil moisture capacity may increase volatilization (4). Soil half-lives from 35 days (3) to 65 days at 15 degrees C or 30 days at 29 degrees C (4) have been reported. Degradation rates are affected by microbial activity and soil moisture levels (4)."


/Pesticide Management Education Program.

"While fluorescein is non-toxic organic compound, bodily contact with the chemical can lead to acute skin and eye irritations, as well inflammation of the digestive and respiratory tracts. It could also potentially cause anaphylactic shock."

I hope the little fishies are ok.